Abstract

Overview

Multiple simultaneous exposures to electromagnetic signals induced adjustments in mammal nervous systems. In this study, the focus was on the non-thermal SAR (Specific Absorption Rate) effects in rat brains exposed to electromagnetic fields (EMF).

Methodology

Forty Sprague Dawley rats were divided into four groups of ten. Each group either exposed or not exposed to EMF radiation in a GTEM chamber for one or two hours. Post-exposure, the ChELISA technique was utilized to assess cellular stress levels and caspase-3-dependent pre-apoptotic activity in both cerebral and cerebellar hemispheres.

Findings

• Significant increases in heat shock proteins (HSP) 90 and 70 were observed 24 hours after exposure, primarily in the cerebral cortex at high SAR levels.
• In the cerebellar hemispheres, single RF and high SAR exposure also showed significant changes in HSP 90, 70, and caspase-3 levels compared to control groups.
• The results suggest that the biological effects of combined EMF signals are significant though not additive, indicating distinct underlying mechanisms.

Conclusion

The study indicates potential health risks associated with non-thermal EMF exposure in terms of inducing cellular stress and pre-apoptotic conditions in mammal brains, specifically highlighting the risks associated with high SAR levels experienced during simultaneous multiple-signal exposures.