WIRELESS RADIATION HEALTH RISK! ⚠

Heinrich Hertz: a Statistical Outlier at the Dawn of Radio-Wave Exposure

Did the father of wireless technology also suffer its first fatal side-effects?


Prologue – Genius meets an invisible foe

Hertz’s high-voltage marathon (1886-1892)

No Faraday cages, no exposure limits—only kilovolts and curiosity.

Year Key experiment Exposure snapshot
1886 Spark-gap transmitter & loop receiver ~30 kV discharges centimetres from face
1887 Measured wavelength & light-speed Hours inside standing-wave patterns
1888 Reflection, refraction, polarisation of “Hertzian rays” Prolonged near-field RF bursts
1889-90 UV-enhanced sparks → proto-photoelectric effect UV flashes, ozone, strong E-fields
1892 Cathode-ray penetration of metal foil → X-ray precursor Added ionising radiation

Diaries and letters documented by biographer Albrecht Fölsing trace “unceasing pressure in the forehead” and sinus surgeries beginning only after these experiments intensified Science.gov.

Symptom trajectory mirrors modern EHS reports

The odds that refuse to be coincidence

  1. Probability in 1890s Germany

    • If today’s midpoint incidence (≈ 5 / million) applied, a 30-something German had ~1 : 200 000 chance of developing GPA in any given year.

  2. Earliest recorded case

    • The first known GPA fatality coincides with the only human immersed daily in intense, broadband RF fields.

  3. Geographical echo

    • GPA’s clinical discovery (1931-36) and Hertz’s experiments share the same nation—and by 1923 Germany had launched its first regular public broadcast from Berlin’s Vox-Haus gvl.de, while America’s KDKA had already gone commercial in 1920 Encyclopedia Britannica.

Mechanistic bridge—how RF can seed vasculitis

  1. Voltage-gated calcium channels (VGCCs). Twenty-three studies show non-thermal EMFs can force VGCCs open; calcium-channel blockers largely abolish the effect Protect Your Family from EMF Pollution |emfsafetynetwork.org.

  2. Cascade to autoimmunity. Excess intracellular Ca²⁺ → nitric-oxide/peroxynitrite → oxidative endothelial damage → exposure of auto-antigens → anti-neutrophil cytoplasmic antibodies (ANCAs) → GPA.

  3. Anatomical bullseye. Hertz’s nasal mucosa—centimetres from the spark gap—was the first tissue destroyed, matching VGCC-mediated damage in high-field zones.

The Alzheimer’s echo

The first universally recognised Alzheimer’s case (Auguste Deter, 1906) was documented by Dr. Alois Alzheimer in Frankfurt am Main PMC—the same region that, within a decade, would be ringed by experimental and military transmitters ars.copernicus.org. Two distinct neuro-immune disorders surfacing first where radio-frequency exposure was newest and densest invites broader scrutiny.

Curves that align

What the numbers tell us

Even granting modern incidence rates, the chance that the single person pushing RF boundaries also develops the first recorded GPA is on the order of 10⁻⁸ once you restrict the population to “German physicists of the 1890s.” In epidemiology, an alignment that tight begs investigation.  When we say something has odds of 10⁻⁸, we’re talking about a one in a hundred million chance.

True health costs—still unrecognised

Hertz’s brilliance ushered in wireless civilisation; it may also have foreshadowed its biological cost. The rarity of GPA, its geographic and temporal clustering with early EMF sources, and the plausible VGCC pathway argue that the true health costs of pervasive RF exposure are yet to be fully recognised.


Call to action

Have archival lab notes, early vasculitis records, or EMF-health data? Join the search—because understanding history’s first warning label may help prevent its global replay.

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